Category: Covid-19

Sydney, Australia:

Covid-19 is still driving Australia's above-average mortality rate, research showed on Monday, with experts predicting the disease's impact will continue to be felt for years to come.

The Australian Actuaries Institute found that five per cent more people died than would be expected in 2023, amounting to 8,400 excess deaths.

Experts measured the number of people who had died as a direct or indirect result of Covid and found the excess mortality rate was still higher than pre-pandemic levels.

About 4,600 deaths were directly attributed to Covid-19, the ninth leading cause of death last year.

Non-Covid-19 excess deaths, meanwhile, were "particularly apparent" in Australians over 75 years old -- with previous infections and interruptions in healthcare related to Covid increasing risks related to heart disease, stroke, diabetes and dementia.

Delays in routine or emergency care and undiagnosed Covid-19 were cited as possible reasons for the higher-than-usual death rate.

Still, the excess mortality rate was lower in 2023 than it was in 2022, according to experts.

"It's encouraging that each successive Covid-19 wave has, so far, resulted in fewer deaths than the previous one," Actuary Institute spokeswoman Karen Cutter said.

"However, we think that Covid-19 is likely to cause some excess mortality for several years to come, either as a direct cause of death or a contributing factor to other causes such as heart disease."

She added the "new normal" level of mortality was likely higher than it would be had the pandemic not occurred.

The report also found Australia's excess mortality was lower than the global average -- with Ecuador, Mexico and Russia showing the highest rates of unexplained deaths.

New Zealand had the lowest excess mortality rate of the 40 countries for which data was available.

(Except for the headline, this story has not been edited by NDTV staff and is published from a syndicated feed.)

Delhi Coaching Centre Deaths: Cops Send Notice To Civic Body

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Covid-19 Still Driving High Mortality Rate In This Country - NDTV

Researchers have just discovered a process in fruit flies which links inflammation with impaired motor function, providing researchers with a potential target for treating the persistent muscle fatigue that follows many infections.

Of long COVID's numerous symptoms, an intolerance to exertion could be considered one of the more debilitating.

"This is more than a lack of motivation to move because we don't feel well," says Washington University developmental biologist Aaron Johnson. "These processes reduce energy levels in skeletal muscle, decreasing the capacity to move and function normally."

With every new infection of the SARS-CoV-2 virus, our risk of experiencing long COVID increases. Almost 18 million adult Americans have now faced this lingering malaise and its exhausting physical symptoms.

Many of these symptoms are familiar, including the frustrating loss of energy that hits around half of all long COVID sufferers. Muscle fatigue is also present in other post-viral conditions, as well as in people suffering from neurodegenerative diseases like Alzheimer's and Parkinson's.

The thing all these conditions have in common is inflammation of our central nervous system. Chemical markers associated with brain injuries have also been identified in COVID patients.

So Washington University developmental biologist Shuo Yang and colleagues used animal models to explore how inflamed neurons can lead to malfunctioning muscles. They identified a signaling pathway between the brain cells and muscles in both flies and mice that leads that leads to a loss of muscle function.

"Flies and mice that had COVID-associated proteins in the brain showed reduced motor function the flies didn't climb as well as they should have, and the mice didn't run as well or as much as control mice," explains Johnson.

"We saw similar effects on muscle function when the brain was exposed to bacterial-associated proteins and the Alzheimer's protein amyloid beta. We also see evidence that this effect can become chronic. Even if an infection is cleared quickly, the reduced muscle performance remains many days longer in our experiments."

In humans, inflammation causes neurons to release the immune cytokine interleukin-6 (IL-6). The team found a comparable protein in their test animals traveled to their muscles via the bloodstream and activated a cellular program called JAK-STAT. JAK-STAT then turned down the amount of energy produced by the muscle tissues' mitochondria power plants.

"We're not sure why the brain produces a protein signal that is so damaging to muscle function across so many different disease categories," says Johnson.

"If we want to speculate about possible reasons this process has stayed with us over the course of human evolution, despite the damage it does, it could be a way for the brain to reallocate resources to itself as it fights off disease. We need more research to better understand this process and its consequences throughout the body.

Yang and team then used drugs to block this pathway in flies to confirm the process can be reversed, as has been shown in previous mouse studies. IL-6 inhibitors have already successfully been used to treat autoimmune diseases such as rheumatoid arthritis, and have shown promise in a few severe COVID-19 cases so far.

"It seems likely that the brain-muscle axis is activated by respiratory infections via the CSF [cerebrospinal fluid] and continues to signal long after the initial infection is cleared," the researchers write in their paper. "Long-COVID may therefore be caused by chronic cytokine signaling."

Some parts of this puzzle remain unclear, the researchers caution, like how SARS-CoV-2 gets into the central nervous system in humans to trigger this inflammation. But this new information could lead to some much needed relief for those suffering from a range of chronic conditions.

By altering chemicals secreted by our neurons, it is now clear how brain inflammation caused by many different conditions can have such a profound physical impact on our entire bodies.

This research is published in Science Immunology.

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We May Have Found a Target For Treating The Fatigue of Long COVID - ScienceAlert

NANTERRE, France (AP) Less than 24 hours after claiming a swimming silver medal at the Paris Olympics, British star Adam Peaty tested positive for COVID-19.

The British Olympic committee announced Peatys condition on Monday, saying he first began feeling ill a day earlier ahead of the 100-meter breaststroke final.

The two-time defending Olympic champion in that event, Peaty just missed out on a third straight gold when he touched two-hundreds of a second behind the winner, Italys Nicolo Martinenghi. Peaty tied for the runner-up spot with American Nic Fink.

In the hours after the final, his symptoms became worse and he was tested for COVID early on Monday morning, Team GB said in a statement. He tested positive at that point.

The 29-year-old Peaty hopes to recover in time to take part in the relays, the statement said. The mixed 4x100 medley relay is set for Friday, while the mens 4x100 medley relay will be held Saturday and Sunday.

As in any case of illness, the situation is being managed appropriately, with all usual precautions being taken to keep the wider delegation healthy, Team GB said.

AP Summer Olympics: https://apnews.com/hub/2024-paris-olympic-games

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British swimming star Adam Peaty tests positive for COVID after winning silver medal - The Associated Press

British Olympic swimmer Adam Peaty has tested positive for COVID-19 at the Paris Games, hours after winning the silver medal in the mens 100m breaststroke final Sunday.

Peaty, now a six-time Olympic medalist, began feeling unwell on Sunday before the event, and his symptoms became worse afterward, the British Olympic Association said in a statement Monday.

In the hours after the final, his symptoms became worse and he was tested for Covid early on Monday morning. He tested positive at that point, the British Olympic Associations statement read, according to The Guardian.

The British Olympic Association added Monday that Peaty is hopeful to be back in competition for the relay events later in the swimming programme [sic].

As in any case of illness, the situation is being managed appropriately, with all usual precautions being taken to keep the wider delegation healthy, the statement continued.

Peaty was seeking a three-peat in the mens 100m breaststroke at the Paris Games following gold medal wins at the 2016 Rio Olympics and the Tokyo Games in 2021.

He was edged out by Italys Nicol Martinenghi by 0.02 seconds.

I gave my absolute all there and I executed it as well as I could, Peaty said, per Team GB.

It doesnt matter what the time says on the board, I know that in my heart Ive already won.

Peaty, who previously took a break from the sport to focus on his mental health, added Sunday that he wasnt crying because of a second-place finish, but because it took so much to get here.

Everything Ive done to this point has happened for a reason and Im so happy that I can race the best in the world and still get joint second, the 29-year-old said.

In my heart Ive won and these are happy tears because I said to myself that I would give my absolute best every single day and I have.

Nic Fink of Team USA placed third in Sundays event.

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British swimmer Adam Peaty tests positive for COVID after Paris Olympics win - New York Post

Just hours after winning silver in the mens 100 breaststroke, Adam Peaty of Great Britain has tested positive for COVID-19, Team GB sent out in a statement Monday afternoon.

Adam Peaty began feeling unwell on Sunday, ahead of his Mens 100 breaststroke final. In the hours after the final, his symptoms became worse and he was tested for COVID early on Monday morning. He tested positive at that point, Team GB said in their release.

He is hopeful to be back in competition for the relay events later in the swimming program. As in any case of illness, the situation is being damaged appropriately, with all usual precautions being taken to keep the wider delegation healthy.

Peaty was the two-time defending Olympic Champion and was in search for a Three-Peaty on Sunday night. He came up just 0.02 seconds short, swimming a 59.05 for silver, tying with Nic Fink of the US. The two went on to share the podium step together.

His time in finals was slightly off his time from semifinals as he led the way through semifinals swimming a 58.86, a time that would have won gold Sunday night as Nicolo Martinenghi charged home to touch first in a 59.03.

Peaty is the World Record holder in the mens 100 breast as his best time stands at a 56.88 from 2019.

He was the fastest swimmer for Great Britain in the 100 breast by over half a second as James Wilby did not advance to the final as he swam a 59.49 in semifinals for 11th.

The good news for the British relay is that the mixed medley relay and mens medley relay do not occur until the end of the meet. Prelims of the mixed medley are scheduled for August 2nd with finals on August 3rd. The mens 4100 medley relay has prelims scheduled for August 3rd and the final on August 4th, the final night of competition.

This is not the first case of COVID for athletes in the village at the Paris Games as Australia had five members of its womens water polo team test positive last week, before the Games even began.

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Adam Peaty Tests Positive For COVID Just Hours After Silver In 100 Breaststroke - SwimSwam

With rising case numbers, COVID-19 seems to be everywhere right now. But did you know the virus might also be literally lurking in your back yard? A new study from scientists at Virginia Tech found SARS-CoV-2 in six of Virginias most recognizable animal species, many of which can be found across the United States.

Weve known for a while that a COVID-afflicted human can pose a risk to their pets. Two domestic cats were the first pets to test positive for the virus in the US way back in early 2020, and the most recent guidance from the Centers for Disease Control and Prevention (CDC) suggests that dogs, hamsters, and ferrets could also be at risk.

Numerous wild species and others held in zoos and sanctuaries have also been reported to be infected, leading the team at Virginia Tech to question just how widespread this virus could be within the local fauna.

This study was really motivated by seeing a large, important gap in our knowledge about SARS-CoV-2 transmission in a broader wildlife community, explained Assistant Professor of Biological Sciences Joseph Hoyt, one of the corresponding authors, in a statement.

They sampled 23 species and detected SARS-CoV-2 RNA in six of them: the deer mouse, Virginia opossum, raccoon, groundhog, Eastern cottontail rabbit, and Eastern red bat. A total of 798 nasal and oral swabs were collected between May 2022 and September 2023, from either wild-caught animals that were later released or animals that were being treated in rehabilitation centers. A further 126 blood samples were also collected from six species.

The researchers wanted to compare animals found in areas with different levels of human contact, from remote rural locations to right in our back yards. The data suggests that the virus has spread into wildlife populations in places with high human activity.

The virus can jump from humans to wildlife when we are in contact with them, like a hitchhiker switching rides to a new, more suitable host, said co-corresponding author and Professor of Biological Sciences Carla Finkielstein. The goal of the virus is to spread in order to survive. The virus aims to infect more humans, but vaccinations protect many humans. So, the virus turns to animals, adapting and mutating to thrive in the new hosts.

We often speak about the risks of viruses spilling over from animals into the human population not least when discussing the origins of COVID itself but rarely do we stop to think about the opposite scenario. A recent study found that viruses jump from humans to animals more often than the other way round, and now this data is showing that we have helped spread SARS-CoV-2 into more animal species than we might think.

I think the big take home message is the virus is pretty ubiquitous, said first author Amanda Goldberg.

Its not clear exactly how the animals caught the infection from humans, but the authors believe that garbage and discarded food are the most likely sources. Two of the mice sampled from the same location on the same day during the study were found to be infected with the exact same variant of the virus, meaning they either caught it from the same human or one infected the other.

Understanding how the virus is spreading in different animal populations is important to gain a more detailed picture of how it is evolving. How might it be spreading from one species to another? Which wild species might serve as reservoirs for human-pathogenic strains?

The black "root" of this evolutionary tree is the original Wuhan SARS-CoV-2 sequence isolated at the end of 2019. Arranged on the tree are 90 different virus sequences isolated in humans since then, and highlighted in red are nine wild animal isolates from species included in this study.

Image credit: figure courtesy of Carla Finkielstein/Virginia Tech

We understood the critical importance of sequencing the genome of the virus infecting those species, said Finkielstein, praising the work of the multidisciplinary team who came together for this study. One result, from an opossum, revealed virus mutations that have previously been unreported, underlining the wealth of useful data that these types of studies can provide.

Finkielstein said, The virus is indifferent to whether its host walks on two legs or four. Its primary objective is survival, adding that SARS-CoV-2 is not only a human problem.

The study is published in Nature Communications.

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COVID-19 Virus Found In 6 Backyard Animals And Humans Are The Cause - IFLScience

Former Hollywood movie mogul Harvey Weinstein, who is awaiting retrial on rape charges in New York, has been hospitalized again.

New York Corrections Department records show that the 72-year-old was transferred from the Rikers Island jail complex in to a locked ward at Bellevue Hospital.

At Bellevue, he is undergoing treatment for COVID-19, double pneumonia and other health conditions, including diabetes, high blood pressure, spinal stenosis and fluid on his heart and lungs, his representative told the Associated Press on Thursday.

We continue to express our gratitude to the officers, doctors and nurses in the citys corrections and public hospital systems who saw to it that Mr. Weinstein was immediately transferred to the Bellevue Hospital Prison Ward, said his publicist, Juda Engelmayer, who attributed Weinsteins health information to his prison consultant, Craig Rothfeld.

Engelmayer did not immediately respond Friday to The Times request for comment.

Weinstein was previously admitted to Bellevue in late April after an appeals court overturned his 2020 rape conviction.

In May, prosecutors asked for a September retrial during a hearing at a Manhattan courthouse. Weinstein had previously appeared in court using a walker, but appeared at the May preliminary hearing in a wheelchair pushed by a court officer. Defense attorney Arthur Aidala said Weinstein was attending the hearing despite his hospitalization and was undergoing unspecified tests due to health issues.

Aidala added that he had no concerns about his clients mental abilities, describing Weinstein as sharp as a tack. As sharp as he ever was, the AP reported.

Reports later surfaced that alleged the Shakespeare in Love and Pulp Fiction producer was receiving special treatment at Bellevue, and he was pulled out of the medical center.

Weinstein was initially sentenced in 2020 to 23 years in prison in New York after allegedly assaulting former production assistant Mimi Haley and once-aspiring actor Jessica Mann. The state appellate court overturned the conviction last spring, determining that the judge who presided over Weinsteins 2020 trial prejudiced his case by allowing four women who accused Weinstein of assault to serve as witnesses despite their allegations not being part of the case.

The court ruled that the trial judge also erred in ruling that prosecutors could cross-examine Weinstein about uncharged and decades-old allegations if he decided to testify.

Weinsteins New York retrial hinges on whether the women who accused him of assault are willing to testify again. Haley has said that she is still considering it, and prosecutors have said Mann is prepared to testify, suggesting setting a date for a retrial after Labor Day.

Weinstein is due to return to court Aug. 7, jail records said.

The disgraced studio boss was convicted of rape in Los Angeles in 2022 and is still sentenced to 16 years in prison in California.

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Harvey Weinstein hospitalized in New York with COVID-19 and double pneumonia - Los Angeles Times

ROCHESTER Wastewater data shows an uptick in COVID-19 infections in many parts of the U.S., including Rochester, this July.

"It definitely seems that COVID case counts are rising both nationally and locally," said Dr. Priya Sampathkumar, an internal medicine physician and head of Infection Prevention and Control at Mayo Clinic.

Data from WastewaterSCAN shows a rising level of COVID RNA found in Rochester's wastewater since late June. The amount of virus detected had been low and stable through the early summer. Now, the level detected mirrors the amount of virus found in late January 2024, as a spike of winter cases fell.

That has translated to primary care providers seeing more patients lately with respiratory illness symptoms, Sampathkumar said.

"(They are) also getting more people who are letting us know that they tested positive at home," Sampathkumar said of her colleagues. "The number of people self-reporting positive tests has definitely gone up in the last couple of weeks."

The number of people hospitalized with COVID has increased slightly this month, Sampathkumar said, but most cases "still seem to be relatively mild, probably because of prior immunity either from vaccination or prior COVID illness."

The percentage of visits to local emergency rooms is still declining, however, said Matthew Giljork, an Olmsted County Public Health epidemiologist.

"Hospitalizations have not translated to an increase in ICU utilization or deaths so far," Giljork said.

Contributed / OMC

At Olmsted Medical Center, they have not observed an increase in hospitalizations, said Dr. Odette El Helou, an infectious disease physician at OMC.

At the state level, per the Minnesota Department of Health's July 25 update, COVID hospitalization rates increased slightly over the previous week but still remain low.

The dominant COVID variant causing infections across the U.S. is KP.3, which Sampathkumar said does not appear to be more transmittable nor cause more severe disease.

A summer wave of COVID infections is not necessarily unusual Sampathkumar said it appears that COVID is settling into a seasonal pattern: a peak of cases in the winter, and a smaller peak in the summer.

"It's probably too early to tell for sure, but it appears at this point that it's settling into sort of a bimodal peak," Sampathkumar said.

Other factors, El Helou said, are people traveling more during the summer and therefore being more exposed to the virus and the level of protection from last fall's COVID vaccines is beginning to wane.

To avoid getting sick, the same precautions that health professionals have been sharing since the start of the pandemic still work.

"Practice good hygiene, wash hands," said El Helou, "good ventilation stay outdoors if possible, open the windows of your home if you have other people in."

Contributed / Mayo Clinic

For vaccinations, the Centers for Disease Control and Prevention last month recommended that everyone 6 months and older receive an updated 2024-2025 COVID vaccine this fall alongside the seasonal influenza vaccine.

"COVID boosters, we hope, will be available by early September," Sampathkumar said. "Everyone should definitely seek out that vaccine and get it. As we've all seen, getting vaccinated may not completely protect you from getting a COVID infection, but it's very, very effective at preventing serious illness and preventing deaths."

If you do get sick, get tested early to see if it is COVID, Sampathkumar said, so you can seek treatment like Paxlovid if you are eligible.

"We recommend you stay home if you have symptoms and that you wear a mask for at least the time that you have symptoms and potentially for up to five days after your symptoms improve to protect everyone else around you," Sampathkumar said.

Contributed / Wastewater SCAN

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Cases of COVID-19 are ticking up in Minnesota. Here's what to know - INFORUM

Since 2020, the condition known as long COVID-19 has become a widespread disability affecting the health and quality of life of millions of people across the globe and costing economies billions of dollars in reduced productivity of employees and an overall drop in the work force.

The intense scientific effort that long COVID sparked has resulted in more than 24,000 scientific publications, making it the most researched health condition in any four years of recorded human history.

Long COVID is a term that describes the constellation of long-term health effects caused by infection with the SARS-CoV-2 virus. These range from persistent respiratory symptoms, such as shortness of breath, to debilitating fatigue or brain fog that limits peoples ability to work, and conditions such as heart failure and diabetes, which are known to last a lifetime.

I am a physician scientist, and I have been deeply immersed in studying long COVID since the early days of the pandemic. I have testified before the U.S. Senate as an expert witness on long COVID, have published extensively on it and was named as one of Times 100 most influential people in health in 2024 for my research in this area.

Over the first half of 2024, a flurry of reports and scientific papers on long COVID added clarity to this complex condition. These include, in particular, insights into how COVID-19 can still wreak havoc in many organs years after the initial viral infection, as well as emerging evidence on viral persistence and immune dysfunction that last for months or years after initial infection.

A new study that my colleagues and I published in the New England Journal of Medicine on July 17, 2024, shows that the risk of long COVID declined over the course of the pandemic. In 2020, when the ancestral strain of SARS-CoV-2 was dominant and vaccines were not available, about 10.4% of adults who got COVID-19 developed long COVID. By early 2022, when the omicron family of variants predominated, that rate declined to 7.7% among unvaccinated adults and 3.5% of vaccinated adults. In other words, unvaccinated people were more than twice as likely to develop long COVID.

While researchers like me do not yet have concrete numbers for the current rate in mid-2024 due to the time it takes for long COVID cases to be reflected in the data, the flow of new patients into long COVID clinics has been on par with 2022.

We found that the decline was the result of two key drivers: availability of vaccines and changes in the characteristics of the virus which made the virus less prone to cause severe acute infections and may have reduced its ability to persist in the human body long enough to cause chronic disease.

Despite the decline in risk of developing long COVID, even a 3.5% risk is substantial. New and repeat COVID-19 infections translate into millions of new long COVID cases that add to an already staggering number of people suffering from this condition.

Estimates for the first year of the pandemic suggests that at least 65 million people globally have had long COVID. Along with a group of other leading scientists, my team will soon publish updated estimates of the global burden of long COVID and its impact on the global economy through 2023.

In addition, a major new report by the National Academies of Sciences Engineering and Medicine details all the health effects that constitute long COVID. The report was commissioned by the Social Security Administration to understand the implications of long COVID on its disability benefits.

It concludes that long COVID is a complex chronic condition that can result in more than 200 health effects across multiple body systems. These include new onset or worsening:

Long COVID can affect people across the lifespan from children to older adults and across race and ethnicity and baseline health status. Importantly, more than 90% of people with long COVID had mild COVID-19 infections.

The National Academies report also concluded that long COVID can result in the inability to return to work or school; poor quality of life; diminished ability to perform activities of daily living; and decreased physical and cognitive function for months or years after the initial infection.

The report points out that many health effects of long COVID, such as post-exertional malaise and chronic fatigue, cognitive impairment and autonomic dysfunction, are not currently captured in the Social Security Administrations Listing of Impairments, yet may significantly affect an individuals ability to participate in work or school.

Whats more, health problems resulting from COVID-19 can last years after the initial infection.

A large study published in early 2024 showed that even people who had a mild SARS-CoV-2 infection still experienced new health problems related to COVID-19 in the third year after the initial infection.

Such findings parallel other research showing that the virus persists in various organ systems for months or years after COVID-19 infection. And research is showing that immune responses to the infection are still evident two to three years after a mild infection. Together, these studies may explain why a SARS-CoV-2 infection years ago could still cause new health problems long after the initial infection.

Important progress is also being made in understanding the pathways by which long COVID wreaks havoc on the body. Two preliminary studies from the U.S. and the Netherlands show that when researchers transfer auto-antibodies antibodies generated by a persons immune system that are directed at their own tissues and organs from people with long COVID into healthy mice, the animals start to experience long COVID-like symptoms such as muscle weakness and poor balance.

These studies suggest that an abnormal immune response thought to be responsible for the generation of these auto-antibodies may underlie long COVID and that removing these auto-antibodies may hold promise as potential treatments.

Despite overwhelming evidence of the wide-ranging risks of COVID-19, a great deal of messaging suggests that it is no longer a threat to the public. Although there is no empirical evidence to back this up, this misinformation has permeated the public narrative.

The data, however, tells a different story.

COVID-19 infections continue to outnumber flu cases and lead to more hospitalization and death than the flu. COVID-19 also leads to more serious long-term health problems. Trivializing COVID-19 as an inconsequential cold or equating it with the flu does not align with reality.

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Long COVID puzzle pieces are falling into place the picture is unsettling - TucsonSentinel.com

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Scientists reveal virus that causes COVID-19 widespread among wildlife - The Elkhart Truth